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Fig. 6 | BMC Ophthalmology

Fig. 6

From: The multimodal imaging features and outcomes of multifocal choroiditis/punctate inner choroidopathy lesion with multiple evanescent white dot syndrome-like features: a retrospective study

Fig. 6

Case 4: First visit (2 weeks after onset) (A-E). Two-month follow-up visit (F-J), and follow-up visit when a CNV was found (K-O). Six-month follow-up visit (P-T). A: Inflammatory cells in the right vitreous body. B: White dots and spots in the macula (blue arrows). C: FAF showed diffuse and scattered hyperautofluorescence in the posterior pole. D: SHRM and the disruption of RPE-BM-CC in the subtemporal fovea on OCT. E: No neovascularization on OCTA. F: Obvious reduction of inflammatory cells in the right vitreous body. G: The yellowish-white lesion shrank. H: FAF showed hypoautofluorescence in the subtemporal fovea. I: OCT showed the heterogeneous SHRM shrank. J: No neovascularization on OCTA. K: Increase of inflammatory cells in the right vitreous cavity. L: The lesion in the subtemporal fovea became larger when oral prednisone was reduced to 10 mg/day. M: FAF showed hypoautofluorescence in the subtemporal fovea. N: Elevation of the SHRM in the subtemporal juxtafovea on OCT. O: The neovascularization was visible on OCTA (red arrow). P: Significant reduction of inflammatory cells in the right eye. Q: The yellowish-white lesion in the right eye shrank with pigmentation. R: FAF showed hypoautofluorescence in the subtemporal macula. S: The SHRM shrank on OCT. T: The neovascularization subsided on OCTA

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BMC Ophthalmology

ISSN: 1471-2415

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