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Fig. 1 | BMC Ophthalmology

Fig. 1

From: RPE disruption and hyper-transmission are early signs of secondary CNV with punctate inner choroidopathy in structure-OCT

Fig. 1

The fundus structure change of a patient had PIC without CNV. (a) Color fundus photography showed sporadic, small, round, yellow-white spots limited to the posterior pole of left eye and no hemorrhage (white arrow). (b) SD-OCT showed the presence of multiple punctured lesions around the macular fovea, and the EZ band of the lesion temporal to the fovea was blurred with hyper-reflective nodule (white arrow). RPE and BM were integral and neither choroidal hyper-transmission nor hypo-transmission was observed below the lesion. (c, d) OCTA angiography en face and B-scan showed no sign of neovascularization around the hyper-reflective nodule (white arrow). (e) OCTA structure en face showed a round-like, elevated lesion in the macular fovea (white arrow), surrounded with multiple punctured lesions with different sizes. (f) Twelve weeks later, the punctate elevated lesion in the macular fovea shrunk (white arrow). (g) SD-OCT showed hyper-reflective material under EZ shrunk and choroid maintained normal transmission (white arrow). (h, i) OCTA angio-Plex and B-scan showed no sign of CNV development during twelve weeks follow-up (white arrow). (j) OCTA structure en face showed a round-like, elevated lesion in the macular fovea (white arrow), which was smaller compared to (e)

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